Source: http://www.thehollywoodgossip.com/2013/04/hand-engraved-honda-civic-coolest-car-vandalism-ever/
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Three years ago, the feds created an online portal at HealthCare.gov to help folks, especially the uninsured, navigate the muddy and mushrooming marketplace of health-care plan options.
While the site is seeing successes, some bugs must be worked out so consumers can obtain complete and accurate information about benefits and prices, according to a report released this week by the inspector general for the U.S. Department for Health and Human Services.
The office found these gaps, among others:
* Insurance products and plans showcased on the website?s ?plan finder? aren?t always available for sale. Some offerings weren?t even recognized by representatives of insurance operators contacted by the inspector general. If the products were available, almost 20 percent of the data displayed didn?t match details provided by insurance operators.
* While most private insurers reported detailed benefit data to the plan finder, as required under the Affordable Care Act, the U.S. Centers for Medicare & Medicaid Services didn?t follow up with insurers that failed to furnish the information. The agency also still hasn?t identified the full universe of providers required to report data.
CMS officials generally agreed with the report, the inspector general said, and are taking taking steps to close the gaps.
You can?follow me?or our?investigative reporting team?on Twitter. We?re also on?Facebook.
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FILE - This undated handout publicity photo shows, from left, Betty White, Valerie Harper, Ed Asner, Mary Tyler Moore, Gavin MacLeod, Cloris Leachman and Georgia Engel, the original cast of the "Mary Tyler Moore Show." White reunites with her former co-stars, Mary Tyler Moore, Cloris Leachman, Georgia Engel and Valerie Harper for an upcoming episode of "Hot in Cleveland." (AP Photo, File)
FILE - This undated handout publicity photo shows, from left, Betty White, Valerie Harper, Ed Asner, Mary Tyler Moore, Gavin MacLeod, Cloris Leachman and Georgia Engel, the original cast of the "Mary Tyler Moore Show." White reunites with her former co-stars, Mary Tyler Moore, Cloris Leachman, Georgia Engel and Valerie Harper for an upcoming episode of "Hot in Cleveland." (AP Photo, File)
This undated publicity photo released by courtesy of TV Land shows, from left, Wendie Malick, Betty White, Valerie Bertinelli and Jane Leeves from TV Land's "Hot in Cleveland." White reunites with her former co-stars, Mary Tyler Moore, Cloris Leachman, Georgia Engel and Valerie Harper for an upcoming "Cleveland" episode. (AP Photo/TV Land)
In this April 4, 2013 publicity photo released by courtesy of TV Land shows, from left, actresses Valerie Harper, Cloris Leachman, Mary Tyler Moore, Betty White and Georgia Engel, former co-stars of the '70s TV classic "The Mary Tyler Moore Show," posing after a press conference discussing their reunion for an upcoming episode of sitcom "Hot in Cleveland," in Los Angeles. (AP Photo/Courtesy TV Land, Danny Feld)
LOS ANGELES (AP) ? It turns out "The Last Show" wasn't the last, after all.
Just around the corner from the intersection of Mary Tyler Moore Avenue and Newhart Street at CBS Studio Center, actress Mary Tyler Moore and some of her former colleagues gathered Thursday afternoon for a press conference to discuss their first sitcom appearance together since "The Last Show," the title of the 1977 series finale of the groundbreaking "The Mary Tyler Moore Show."
Moore, Cloris Leachman, Betty White, Georgia Engel and the ailing Valerie Harper reunited this week to tape an episode of White's hit TV Land comedy, "Hot in Cleveland."
On "The Mary Tyler Moore Show," Harper played the free-spirited best friend Rhoda Morgenstern opposite Moore's prim Mary Richards.
Harper, 73, wondered if the group had come together again because of her recent diagnosis with terminal brain cancer.
"Do you guys think this was part of my condition?" asked Harper.
"Oh, yes. I do," replied co-star Leachman, who portrayed Mary and Rhoda's snooty apartment-building manager, Phyllis Lindstrom. "They had the idea and they wrote this amazing script," Leachman continued. "We're just almost in tears, we're so thrilled."
Harper recently told the "Today" show that in January she was diagnosed with a rare brain cancer and told she had three months to live. But, on this day, Harper was robust. "I'm kind of symptomless for this particular condition, so we're just going to see," she explained.
Harper said this reunion has been more about laughter than tears, in spite of her diagnosis. "And the tears come up, but they're part of the laughter," she noted. "Did you ever laugh 'til you cry?"
This group has reunited before, in 2007 to present at the Screen Actors Guild Awards and for a 2008 episode of "The Oprah Winfrey Show." But this is the ensemble's first acting job together in some 36 years. Coincidentally, it's taking place on the same studio lot their Emmy-winning series was filmed between 1970 and 1977.
The actresses won't be playing their "Mary Tyler Moore Show" characters on the "Hot in Cleveland" episode, which is scheduled to air Sept. 4. The storyline has Elka (White) and Mamie Sue (frequent guest star Engel) reuniting with their long-lost bowling teammates: Diane (Moore), Peg (Leachman) and Angie (Harper). While the characters names are different, each will provide a nod to the roles the actresses played on "The Mary Tyler Moore Show."
"(Rhoda) taught me to thank your lucky stars for a fabulous friend," Harper noted, referring to Mary Richards and pointing to Moore and laughing.
Both the first "Mary Tyler Moore Show" and the new "Hot in Cleveland" episodes are titled "Love is All Around," and that seemed to be the case at this press conference.
"We don't have to fake it," Moore said.
____
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“Buckwild” Paying For Shain Gandee Funeral As 911 Call Released (AUDIO)
The producers of the MTV reality show “Buckwild” are taking care of the expenses of Shain Gandee’s funeral, following the tragic death of the reality star of carbon monoxide poisoning. The news comes as the heart-breaking 911 call was released, after a friend located the three men’s lifeless bodies in the Ford Bronco. An autopsy ...
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Jill Wills
A recent survey by the Pew Internet and American Life project found that 72% of Americans experience some form of dropped calls, and 32% experience dropped calls at least a few times per week or more. Like it or not, dropped calls are still a major problem for a large number of people, and while the carriers are continuing to add towers and expand coverage areas, this is not a problem that's going to go away soon. So what is there to do?
After years of research into this problem, the FCC formally approved the use of cell phone signal boosters to extend the range of cell networks into areas that receive poor service, including homes, offices, commercial buildings and more. We'll take a closer look at what cell phone signal boosters are and give some guidance on how to choose the right system to permanently solve your reception problems.
When it comes to poor cell phone signal, there are two main contributing factors: distance from the closest cell tower and obstructions causing interference.
Carriers provide cell coverage through a network of strategically placed cellular towers. Ideally, whenever you're within this network, your mobile device will automatically connect with the closest tower, and as you move, will continue hopping to the next closest tower. Unfortunately, as you get towards the edge of your carrier's coverage area, there won't be new towers to hop to, and your mobile device will start to experience degraded signal the further it is from the nearest tower. You'll reach a point where your mobile device is too far from the tower to maintain a consistent connection, and you'll experience dropped calls.
While cell signal passes through the air with minimal trouble, every object it meets between the tower and your mobile device causes some degree of interference. Geographical elements, such as hills, mountains, and trees can cause major signal issues, as well as building materials, like metal siding, concrete, and wire mesh. Additionally, the increased use of energy efficient products, such as radiant barriers and window tinting, have contributed to the degree of cell phone reception problems in new homes.
While either one of these factors are enough to cause dropped calls and poor reception, the combination of the two all but guarantees problems. Next we'll look at how a cell phone signal booster is designed to deal with both of these factors.
Cell phone signal boosters (also known as cell phone repeaters) were developed to address the two factors that cause poor cell phone reception. They're designed to amplify a weak outside signal and bypass any obstructions to provide a strong inside signal to an area that was originally lacking.
The system works by mounting an outside antenna in a location that currently has signal, which is typically on the roof. The signal is passed from the outside antenna, by a cable to a signal amplifier inside the building. Once the signal is amplified, it is then sent to an inside antenna, where it is broadcast out to the area which needs better reception. The system also works in reverse, with the signal from your phone being amplified and broadcast back to the tower, ensuring strong, two-way communication.
In order to select the correct cell phone signal booster for your situation, you need to know the following three pieces of information: The carriers & networks that you need to support, the existing outside signal strength of those carriers, and the size of the area that needs to be covered in boosted signal. Let's review those in more depth:
Cell phone signal boosters only amplify specific frequencies of radio waves, which pertain to specific carriers (AT&T, Verizon, Sprint, etc.) and networks on those carriers (2G, 3G, or 4G LTE). The first thing you need to know is which carrier(s) you need to support, as well as which networks on those carriers, as that determines which signal amplifiers you'll be able to use and which type of outside antenna you'll need.
Most North American carriers (except for T-Mobile and Nextel) use the same frequencies for 2G and 3G networks (which handle voice calls and 3G data), so the same signal amplifier can be used to amplify all of those carriers at the same time. The 4G LTE networks (which currently only handle fast data, no voice) on each carrier use different frequencies, so if you want to boost 4G LTE, then you'll need an amplifier that is specifically designed for that carrier's 4G LTE network.
In addition, if you need to support multiple carriers, then you'll want to use an omnidirectional (omni) outside antenna, which can send and receive from all directions at once. If you only need to support one carrier, then you can use a stronger yagi directional antenna, which you can aim at the closest cell tower and receive more boosting power than you would receive from an omni antenna.
The outside signal strength of the carriers you need to support determines how strong of an amplifier you need to provide adequate coverage for the area you need covered. The weaker the outside signal is, the stronger the cell phone signal booster needs to be in order to boost the signal and provide adequate coverage.
The last piece of information that will determine which type of signal booster you need is the size of the area that needs to be covered in boosted signal, as well as the layout of the area. The combination of outside signal strength and the size of the area to be covered will determine how strong of an amplifier you'll need.
In addition, the layout of the area to be covered will determine which type of inside antenna you'll need. If the entire coverage area is located on one floor, then a dome antenna will be the best choice for distributing the boosted signal, but if the coverage area is over multiple floors, then you'll want to go with a panel antenna, which is more directional and can be used to ensure signal is distributed across the required area.
Until this point, we've been discussing cell phone signal boosters for buildings, but there are also vehicle signal boosters for cars, trucks, RVs and even boats. While the principles are the same, the outside signal strength and coverage area come less into play, as those are constantly changing as the vehicle moves. Rather, the choice really depends on which carriers and networks you need to support.
Finally, there is a style of signal booster called a cradle, which is meant for one device at a time, and the device must be sitting the cradle the entire time it's in use. It's the most mobile of the signal booster options, as the system consists of only the cradle and a magnetic mount outside antenna, and is also the most affordable, but is less convenient when talking on the phone, as you need to use a Bluetooth headset or speakerphone while in use.
After years of working with both cell phone signal booster manufacturers and cellular carriers in the United States, the FCC announced in February the formal approval for the use of cell phone signal boosters by consumers in the U.S. In addition to this approval, the FCC also instituted checks to ensure that signal boosting equipment will not interfere with carrier networks, which include the registration of a cell phone signal booster with the carrier that it will be used with, as well as the certification of new signal boosters against a stringent set of standards, to ensure that only safe equipment is being sold to the public. These checks are anticipated to go into effect in the fall of 2013. Overall, both manufacturers and carriers regard this as a great step forward to helping consumers receive better service in their homes, offices, and vehicles.
Understanding the basics of how a cell phone signal booster works and what you need to think about when purchasing one is a huge first step in determining the right system for you. Every situation is different, from the local geography to the construction of the house or building, so if you have the opportunity, consult with a signal booster expert as to what system is best for you.
Jill Wills is head of community & customer support at UberSignal, an industry leader in the design and supply of cell phone signal booster solutions. She loves helping people solve their reception problems, and is a regular contributor to the UberSignal blog.
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Apr. 2, 2013 ? A paper published this month in the medical journal The Lancet Neurology suggests that a broad spectrum of developmental and psychiatric disorders, ranging from autism and intellectual disability to schizophrenia, should be conceptualized as different manifestations of a common underlying denominator, "developmental brain dysfunction," rather than completely independent conditions with distinct causes.
In "Developmental Brain Dysfunction: Revival and Expansion of Old Concepts Based on New Genetic Evidence," the authors make two key points:
? Developmental disorders (such as autism and intellectual disability) and psychiatric disorders (such as schizophrenia and bipolar disorder), while considered clinically distinct, actually share many of the same underlying genetic causes. This is an example of "variable expressivity:" the same genetic variant results in different clinical signs and symptoms in different individuals. ? When quantitative measures of neuropsychological and neurobehavioral traits are studied instead of categorical diagnoses (which are either present or absent) and individuals are compared to their unaffected family members, it is possible to more accurately demonstrate the impact of genetic variants.
According to Andres Moreno De Luca, M.D., research scientist at the Autism and Developmental Medicine Institute at Geisinger Health System and article co-author, "Recent genetic studies conducted in thousands of individuals have shown that identical genetic mutations are shared among neurodevelopmental disorders that are thought to be clinically distinct. What we have seen over the past few years is that genetic mutations that were initially found in individuals with one disorder, such as intellectual disability or autism, are then identified in people with an apparently different condition like schizophrenia, epilepsy, or bipolar disorder."
"It turns out that the genes don't respect our diagnostic classification boundaries, but that really isn't surprising given the overlapping symptoms and frequent co-existence of neurodevelopmental disorders," said Scott M. Myers, M.D., autism specialist at Geisinger Health System and article co-author.
"We believe this study supports use of the term 'developmental brain dysfunction' or DBD, which would encompass the broad spectrum of neurodevelopmental and neuropsychiatric disorders," said David H. Ledbetter, Ph.D., executive vice president and chief scientific officer at Geisinger Health System, and article co-author. "Additionally, it is clear that diagnostic tools such as whole genome analysis for both children and their families are essential when diagnosing and treating these disorders in order to ensure the most personalized treatment."
An example used in the study was analysis of intelligence quotient (IQ) scores. The average IQ score in the general population is 100. Historically, the medical community has defined intellectual disability as an IQ of less than 70 (with concurrent deficits in adaptive functioning). But according to Dr. Ledbetter, there is little difference in the function of a child with an IQ of 69 versus 71, yet one may be diagnosed with a disability and the other may not.
"We know a variety of factors contribute to IQ score, including genetics, as a child's IQ is highly correlated with that of his or her parents and siblings. Therefore, an important factor to take into consideration when interpreting IQ is family background," said Dr. Ledbetter. "Imagine if we have a child with a genetic abnormality, but the child's IQ is 85. Technically, we would not diagnose this child with a disability. However, if the family of this child has IQs around 130, we could consider that this child's genetic anomaly has 'cost' him or her 45 IQ points -- a very substantial difference."
According to Dr. Myers, "One implication of this concept is that studies designed to investigate the causes and mechanisms of developmental brain dysfunction should focus on measurement of quantifiable neuropsychological and neurobehavioral traits across groups of individuals with different clinical diagnoses. Another is that whenever possible, individuals with a particular genetic variant or other risk factor should be compared to their unaffected family members, not just to population norms."
Other authors on the paper were Thomas Challman, M.D. of Geisinger; Daniel Moreno De Luca, M.D., of Yale University, New Haven, Conn.; and David Evans, Ph.D., of Bucknell University, Lewisburg, Pa.
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